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Turbellaria include mobile bottom-living predatory atworms. The trematodes includes various ukes, such as the tropical schistosome that causes bilharzia; cestodes are tapeworms: both these groups are important parasites of shes and other vertebrates including humans. Molluscs are often important intermediate hosts May be parasitic, herbivorous, or predatory; some parasitic forms reach considerable size. Poorly known; may well be more diverse than is currently recognized Oligochaetes are bottom-living worms that graze on sediments; leeches are mainly parasitic on vertebrate animals, some are predatory Both bivalves and gastropods have speciated profusely in certain freshwater ecosystems. The larvae of many bivalves are parasitic on shes. Because of their feeding mode, bivalves can help maintain water quality but tend to be rather vulnerable to pollution Include larger bottom-living species such as shrimps, cray sh, and crabs of lake margins, streams, alluvial forests, and estuaries. Also larger plankton: lterfeeding Cladocera and lter-feeding or predatory Copepoda. Many isopods and copepods are important sh parasites In rivers and streams, grazing and predatory aquatic insects (especially larval stages of ying adults) dominate intermediate levels in food webs (between the microscopic producers,

Most individuals who experience stress do not develop illness Stressful life changes are usually temporary, whereas other risk factors for disease can be longer lasting, for example, smoking, alcohol consumption, a high-fat, low- ber diet, and risky lifestyle in general When comparing a single life event with those long-term behaviors, the latter seem to be more in uential in developing illness Moreover, the experience of a critical life event is related to coping and social support, whereby these two factors may moderate the stressillness connection How can we understand the mechanisms of the stress-illness association There are three major pathways that link stressful life events to ill health (Figure 22) The main pathway places physiological changes as a mediator between origin and outcome, in particular, changes of immune parameters, and endocrine and cardiovascular reactivity.

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Recent research, for example, in the eld of psychoneuroimmunology, has documented progress in identifying bodily responses to stress that constitute precursors of disease (see Ader, 2001; Herbert & Cohen, 1993a, 1993b) Endocrine and cardiovascular reactivity, as expressed in blood pressure, heart rate, or catecholamine excretion, is considered a stress-based codeterminant of cardiovascular disease, including myocardial infarcts The amount of reactivity is, however, not exclusively governed by the stress experience Rather, it is moderated by genes, personality, age, and gender, as well as other factors (Weidner, 2001) The other major pathway is represented by healthcompromising behaviors People under stress might want to relieve their tension by consuming more tobacco, illicit drugs, alcohol, and so on They feel too absorbed by their stress to monitor their diets and to maintain other preventive behaviors Adherence to routine self-care might suffer during a stress episode.

P1 (X) in probability (11.204) P2 (X) (11.205)

Among smokers, stress may increase the number of cigarettes consumed as well as the intensity of smoking by deep inhaling When under stress, women seem to be more likely to engage in unhealthy eating behaviors, whereas men tend to turn to drinking and illicit drug use (Brannon & Feist, 1997) A third pathway pertains to all kinds of negative affect often associated with experiencing stress Constant rumination, worrying, anxiety, pessimism, depression, and anger are health compromising in the long run Studies have shown that optimism is related to good health, whereas depression can be a precursor of sickness (Carver, 2001) The mechanism of pathogenesis operates through physiological changes, including immune suppression and blood pressure elevations Scheier and Bridges (1995) reviewed depression and health outcomes Depression may be a general risk factor for premature death The evidence for mortality effects is most compelling for cardiac disease Studies indicate that cardiac.

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